
Which of the following statements is false for Angiotensin.
i. It is a steroid hormone.
ii. Angiotensinogen is converted to angiotensin I by renin.
iii. Angiotensin II decreases the release of aldosterone.
iv. Angiotensin II brings about vasoconstriction.
v. It stimulates Na reabsorption from the Proximal convoluted tubule.
A i, ii and iii
B i and iii
C iii, iv and vi
D i, iii and v
Answer
467.4k+ views
Hint: Renin is an enzyme produced by the kidneys (and potentially the placenta) as part of a physiological mechanism that controls blood pressure. Renin interacts with a protein called angiotensinogen in the blood, causing angiotensin I to be released.
Complete answer:
Angiotensin is a peptide hormone that regulates blood pressure. As a result, assertion I is incorrect. Angiotensinogen is converted to angiotensin I by Renin. Angiotensin I is converted to Angiotensin II by the Angiotensin Converting Enzyme. It boosts the production and release of the hormone Aldosterone. As a result, assertion ii is incorrect. Angiotensin II raises blood pressure via causing vasoconstriction and sodium reabsorption from the proximal convoluted tubule.
As a result, the proper response is B. i and iii.'
The peptide hormone angiotensin II is widely known for its endocrine involvement in blood pressure, fluid, and electrolyte homeostasis regulation.
Angiotensin I is converted to angiotensin II by the enzyme angiotensin-converting enzyme (ACE). Angiotensin II binds to certain receptors in the brain, kidney, adrenal, vascular wall, and heart and exerts its actions.
Angiotensin II increases sodium reabsorption via boosting Na-H exchange in the proximal convoluted tubule of the kidney. Increased Na levels in the body cause the blood to become more osmolar, causing fluid to transfer into the blood volume and extracellular space (ECF).
Primary hyperaldosteronism is a hormonal condition in which the adrenal glands generate too much aldosterone hormone, which raises salt levels in the blood, resulting in high blood pressure.
People with chronic obstructive pulmonary disease (COPD) may have lower ACE levels (COPD) Emphysema, lung cancer, and cystic fibrosis are all lung illnesses. Starvation.
Note:-
Furthermore, new research has indicated that ARBs reduce cardiovascular events more effectively than ACE inhibitors, particularly in patients with preexisting cardiovascular disease. ARBs have less side effects than ACE inhibitors; in general, ARBs are better tolerated than ACE inhibitors.
Complete answer:
Angiotensin is a peptide hormone that regulates blood pressure. As a result, assertion I is incorrect. Angiotensinogen is converted to angiotensin I by Renin. Angiotensin I is converted to Angiotensin II by the Angiotensin Converting Enzyme. It boosts the production and release of the hormone Aldosterone. As a result, assertion ii is incorrect. Angiotensin II raises blood pressure via causing vasoconstriction and sodium reabsorption from the proximal convoluted tubule.
As a result, the proper response is B. i and iii.'
The peptide hormone angiotensin II is widely known for its endocrine involvement in blood pressure, fluid, and electrolyte homeostasis regulation.
Angiotensin I is converted to angiotensin II by the enzyme angiotensin-converting enzyme (ACE). Angiotensin II binds to certain receptors in the brain, kidney, adrenal, vascular wall, and heart and exerts its actions.
Angiotensin II increases sodium reabsorption via boosting Na-H exchange in the proximal convoluted tubule of the kidney. Increased Na levels in the body cause the blood to become more osmolar, causing fluid to transfer into the blood volume and extracellular space (ECF).
Primary hyperaldosteronism is a hormonal condition in which the adrenal glands generate too much aldosterone hormone, which raises salt levels in the blood, resulting in high blood pressure.
People with chronic obstructive pulmonary disease (COPD) may have lower ACE levels (COPD) Emphysema, lung cancer, and cystic fibrosis are all lung illnesses. Starvation.
Note:-
Furthermore, new research has indicated that ARBs reduce cardiovascular events more effectively than ACE inhibitors, particularly in patients with preexisting cardiovascular disease. ARBs have less side effects than ACE inhibitors; in general, ARBs are better tolerated than ACE inhibitors.
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